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Erratum to: Cytoplasm-predominant Pten associates with increased region-specific brain tyrosine hydroxylase and dopamine D2 receptors in mouse model with autistic traits
Molecular Autism volume 7, Article number: 14 (2016)
The Original Article was published on 17 November 2015
Erratum
We have just noticed a minor error in Fig. 1a of our article [1]. The m3m4 mutation was described incorrectly as it improperly describes two Pten mutations, R233N and K269N. However, the confirmed sequence data on the m3m4 mutation indicates there are five nucleotide changes, as we have previously published [2], resulting in four amino acid changes: R233Q, R234Q, K265N, and K266Q. The fifth nucleotide change is a synonymous mutation, L264L. For greater clarity on the details of the nucleotide changes and the corresponding amino acid changes of the m3m4 mutation, they have been provided:
a. NLS-like region of Pten and the missense mutations created in the NLS-like region
Additionally, the included Fig. 1a now shows the correct amino acid changes. We apologize for any confusion caused by this error.
Reference
He X, Thacker S, Romigh T, Yu Q, Frazier Jr TW, Eng C. Cytoplasm-predominant Pten associates with increased region-specific brain tyrosine hydroxylase and dopamine D2 receptors in mouse model with autistic traits. Mol Autism. 2015;6:63.
Tilot AK, Gaugler MK, Yu Q, Romigh T, Yu W, Miller RH, et al. Germline disruption of Pten localization causes enhanced sex-dependent social motivation and increased glial production. Hum Mol Genet. 2014;23(12): 3212–27. doi:10.1093/hmg/ddu031.
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The online version of the original article can be found under doi:10.1186/s13229-015-0056-6.
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He, X., Thacker, S., Romigh, T. et al. Erratum to: Cytoplasm-predominant Pten associates with increased region-specific brain tyrosine hydroxylase and dopamine D2 receptors in mouse model with autistic traits. Molecular Autism 7, 14 (2016). https://doi.org/10.1186/s13229-016-0075-y
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DOI: https://doi.org/10.1186/s13229-016-0075-y