Fig. 3From: Cross-level analysis of molecular and neurobehavioral function in a prospective series of patients with germline heterozygous PTEN mutations with and without autism(Left) Western blots on murine brain tissue at 6 weeks of age showing levels of PI3K pathway mediators in a model of PTEN mutation. (Right) Quantification of signal intensity for each mediator normalized to Gapdh (n = 3 each)Back to article page